Neuromyelitis optica spectrum disorder (NMOSD) is an AQP4-IgG or MOG-IgG-mediated astrocytopathy affecting 15,000–20,000 Americans, causing severe relapsing optic neuritis and transverse myelitis via complement-mediated astrocyte destruction, BBB disruption, and secondary demyelination. NMOSD involves IL-6-driven B-cell activation and NF-kB inflammatory signaling. Sea moss fucoidan has anti-complement C1q properties and modulates NF-kB neuroinflammation; selenium supports CNS GPx4 neuroprotection; omega-3 DHA supports myelin integrity; zinc supports BBB ZO-1 tight junction maintenance. Eculizumab, inebilizumab, and satralizumab are standard; sea moss provides neuroprotective nutritional support alongside neurologist care.

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