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Hereditary angioedema (HAE) is a rare life-threatening condition affecting 1 in 50,000 people, caused by C1 inhibitor (C1-INH) deficiency or dysfunction leading to uncontrolled contact activation, bradykinin overproduction, and recurrent episodes of subcutaneous, submucosal, and laryngeal edema. HAE attacks can be life-threatening when laryngeal involvement causes airway obstruction. Sea moss fucoidan has anti-complement C1q/C4 properties and anti-bradykinin-related vascular permeability effects; selenium supports endothelial GPx protection during oxidative edema attacks; zinc supports endothelial tight junction ZO-1/occludin barrier function; omega-3 modulates inflammatory eicosanoids. CRITICAL: HAE attacks require C1-INH concentrate, icatibant, or lanadelumab — sea moss NEVER replaces emergency HAE treatment; always carry rescue medication.
