Sea Moss for POTS: Fucoidan & Autonomic Anti-Inflammatory Support for Postural Tachycardia Syndrome

Postural orthostatic tachycardia syndrome (POTS) is a form of dysautonomia defined by ≥30 bpm heart rate increase (≥40 in <19y) within 10 min of standing without orthostatic hypotension; estimated 1-3 million Americans (F:M ~5:1, peak 15-35y); mechanisms: autoimmune (anti-adrenergic receptor IgG — anti-alpha1-AR, anti-beta1-AR, anti-beta2-AR; anti-muscarinic M3/M4 IgG in subset); neuropathic POTS (small fiber neuropathy, skin biopsy-confirmed, anti-tTG IgG overlap); hyperadrenergic POTS (elevated standing norepinephrine >600pg/mL); post-COVID POTS (autoimmune, S1 spike ACE2); NF-kB/IL-6/IL-17 neuroinflammation; mast cell activation syndrome (MCAS) overlap 30%; hypovolemia + reduced venous return; Ehlers-Danlos syndrome overlap.

Sea moss fucoidan targets NF-kB, IL-6, IL-17, autoimmune antibody inflammation; selenium provides neuronal/autonomic GPx neuroprotection; omega-3 DHA supports autonomic nerve membrane integrity and resolvin D1 neuroinflammation; zinc supports autonomic metalloenzymes and small fiber nerve integrity. Read the full guide at Sea Moss for POTS.

This content is for educational purposes only. Consult your cardiologist or neurologist for POTS diagnosis and management.